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Posterior Third Lameness.
A series of articles by Peter N Baker.
An in depth study of posterior third lameness of the equine front foot.
To a greater degree a predictable condition, which by good farriery, husbandry
and attention to hoof capsular shape can be arrested if not reversed.
Part Three:
14. The Hybrid foot.
The so called hybrid foot will now be considered. One heel will be a
normal open type, or somewhat flat and weak heeled. The other heel will
be driven forwards, and the coronary band above this heel will displaced
upwards and outwards, and may even overhang the vertical plane to the
ground surface of the hoof capsule. The run forwards heel causes massive
pressures on its cartilage, and without efficient farriery husbandry the
result will be a well defined
unilateral side-bone. This foot will be one of a mismatched pair. The
hybrid foot is invariably the one that suffers the bulk of posterior third
lameness problems.
The question is now raised, "What causes two front feet to become
mismatched?" Setting aside the inherited factor, "Club Foot
Syndrome", and especially when it applies to the insidious type of
onset, the answer lies in the unbalanced trauma resulting from the modified
roles of leading and weight bearing, this in turn results in the feet
breaking over at
different stages relative to each other. The lead leg breaks over considerable
earlier than the support leg. The mechanically induced effect of this
seems to cause an open weak flat type of foot to develop on the lead leg,
and an upright more contracted, or a hybrid foot on the support leg, ("Mismatched
Feet"). The predisposing factors relating to the leading leg have
been dealt with previously (sections 3 and 12).
The factors affecting the developing, weight bearing hybrid foot seem
to stem from the increased loading that is placed upon it. The hybrid
foot tends to turn inwards or very occasionally outwards, depending on
the animals conformation, (basewide, base narrow, toe in, toe out), this
unnatural positioning allows the limb to be loaded directly distal to
the
animals balance axis. The result of this modified positioning is that
either the medial or lateral heel will suffer more trauma than its partner.
This heel will be displaced forwards and the proximal area of the coronary
band will be driven upwards. The changes to this foot are further influenced
by its position at break over, relative to the position of the leading
leg at the point where it breaks over, (see above).
In the early stages of the development of upright and hybrid feet trauma
causes a the disruption of the physical structure of the hoof. The author
feels this leads to dramatic changes to the vascularity and blood supply,
and will induce the bar heel laminitis condition which predisposes a low
grade laminitic founder in the heel area of the foot. Radiographs of P3
may show a reverse rotation, a vertical displacement, or a medial / lateral
displacement. This condition differs from the laminitis found in the dorsal
wall condition, in so much as serum pooling and the attendant gas pockets,
are not as radiographically evident, laminal stretching and breakdown
is not so obvious. This masking is probably due to the flexible nature
of the underlying laminal bed, the Lateral Cartilage.
A study of radiographs of a developing hybrid foot, of a horse exhibiting
acute posterior third lameness seem to indicate a backward rotation of
the distal phalanx; the normal 10 degree angle between the base of P3
and the ground is decreased, this condition was further complicated by
a medial / lateral imbalance. The backwards rotation may account for,
a posterior movement of the apex of the extensor process of P3, and a
vertical displacement leading to the formation of two groves seen by,
Duckett Newmarket Symposium 1990 ). The same condition may lead to the
laminal tearing located between the tip of the extensor process and the
proximal inner face of the hoof capsule. Noted by Eustace, ( RDC, Course,
Salisbury 1990 ).
It is worth comparing the base angle in the normal foot and the developing
upright or hybrid foot. In the normal foot the base angle of P3 is about
10 degrees, (The heel being elevated). In the acute stage of the developing
bilateral upright foot, the base angle is decreased, less than 10 degrees,
(reversed rotation). In the chronic stage of the condition, the base angle
is dramatically increased, more than 10 degrees.
The posterior founder state referred to above, and the medial / lateral
imbalance would explain a progression which is seen, the rapid development
of the upright or hybrid foot. The equine physiology is effectively restoring
P3 to a comparative position as it refers to its other front foot.
In both the developing (Acute Stage) upright and hybrid foot, and specifically
as it refers to the insidious type of onset, the soft tissue distal to
P3 undergoes compression, the resulting sensitivity induces a shuffling
gait and the posterior element of the animals stride will be reduced.
The flexor group of muscles seem to change their dynamic function , and
have to lift the limb prior to it becoming fully unloaded, this action
takes place earlier than the point of breakover of the sound animal. This
probably triggers a biomechanical response and an added tendon tension,
the tendon surface of the distal sesamoid and the navicular bursa suffer
trauma. The effect is it causes the animal to unload its heels Transition
Stage,) due to an associated pain. (Heel Sore). Nature now dictates that
the heel must develop in order to contact with the ground. Rapid heel
development is now seen. In the chronic hybrid foot the heel elevation
is not balanced, the contracted heel is displaced forwards, and the horny
covering is different, (CT Swan, current ongoing study, 1997), in so much
that there is a greater length of horn between the hair line at the heel
and the ground surface. The base angle of P3 on this side of the foot
will be greater than its co-lateral heel. This will cause the hoof capsule
to rotate, twisting of the horn tubules will be noticed, and represents
the hoof capsule or part of it rotating on the laminae which cover the
outer surface of the lateral cartilages, and the posterior part of the
distal phalanx. In animals that have suffered prolonged incidence of a
hybrid foot, the distal phalanx undergoes a change in form, and it becomes
twisted (Chronic Stage). A very recent observation also indicates that
in an animal that had this type of foot for many years, the middle and
proximal phalanxes changed their form. In fact a bone specimen in the
care of the author, demonstrates that the upper and lower articular surfaces
of a proximal phalanx were not parallel, this bone is effectively bent.
If nature is allowed to deal with the condition, and especially when
it refers to the shod animal. The systemic jolt which the upright or hybrid
foot receives from this foundered condition seems to go beyond what is
needed to restore P3 to its natural position within the hoof capsule;
the heel or heels spontaneously continue to develop and the limb effectively
becomes longer, (Challoner, Forge Magazine, 1989), than the limb with
the open foot. The application of steel to the bottom of the foot prevents
nature wearing away of surplus horn. Regular and efficient hoof care at
this stage of the syndrome is imperative if chronic changes of hoof form
are to be avoided. At this stage of the condition extreme pressure is
placed upon the lateral cartilages of upright foot, or the cartilage of
the hybrid foot, the hoof wall is now far more vertical, the digital cushion
is no longer effectively retained within the converging angles of the
cartilage within the horny box, and will now become located in an elevated
position , and may be seen to bulge outwards in the coronary area. Under
loading cartilage will dramatically distort, and sometimes is seen to
bulge outwards distorting the hoof wall as previously mentioned, whether
it is in one heel or both heels depends upon if it is a hybrid or upright
type of foot.
This phenomenon accounts for the bulk of lameness associated with the
upright and hybrid type of feet, especially when it relates to the soft
tissue sensitivity. Due to compression from many directions, and loss
of the freedom to move in an unrestricted manner, within the normal confines
of the hoof capsule, a considerable amount of pain will be experienced
by the animal.
We know from our experience with normal dorsal wall laminitis that pedal
bone realignment can only be effected by frog support, The immediate elevation
of a foundered heel, whether unilateral or bilateral, can only be achieved
by the use of a wedge heeled shoe, or a wedged heel pad, and in the long
term, the encouragement of natural heel development by the use of bar
shoes, frog supports and horn hardening chemical compounds. Following
suggestions from fellow farriers, and especially when taking into account
the observations of CT Swan, it seems that at the acute stage when hoof
capsule imbalance is bought about by outside forces or poor husbandry
, the elevation of run forwards contracted heel by the use of a wedged
up heel, may effect an increased degree of soundness, and improvement
in horn quality. Prolonged elevation is found to be counter productive
to the well-being of the affected animal and in practical term short term
use is all that is recommended to effect improved venous circulation,
to improve the rate of hoof heel horn growth.
Dramatic changes to the hoof capsule of a sound equine athlete, with
a chronic hybrid foot, can only be for the cosmetic benefit of a third
party , and will lead to skeletal damage or fracture, as the author unfortunately
found out. In fact that is the reason this part of this article was written.
The next article will look at the progression of posterior third lameness
First published in Forge Magazine 1998. Copyright Peter N Baker.
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